Ca(2+)/Calmodulin-dependent Protein Kinase II Inhibitors Disrupt AKAP79-dependent PKC Signaling to GluA1 AMPA Receptors

Eitemau Tebyg

• AKAP79 Selectively Enhances Protein Kinase C Regulation of GluR1 at a Ca(2+)-Calmodulin-dependent Protein Kinase II/Protein Kinase C Site
  gan: Tavalin, Steven J.
  Cyhoeddwyd: (2008)
• Preferential generation of Ca(2+)-permeable AMPA receptors by AKAP79-anchored protein kinase C proceeds via GluA1 subunit phosphorylation at Ser-831
  gan: Summers, Kyle C., et al.
  Cyhoeddwyd: (2019)
• Ca(2+)/Calmodulin-dependent Protein Kinase II Binds to and Phosphorylates a Specific SAP97 Splice Variant to Disrupt Association with AKAP79/150 and Modulate α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid-type Glutamate Receptor (AMPAR) Activity
  gan: Nikandrova, Yelyzaveta A., et al.
  Cyhoeddwyd: (2010)
• Ca(2+)/Calmodulin Disrupts AKAP79/150 Interactions with KCNQ (M-Type) K(+) Channels
  gan: Bal, Manjot, et al.
  Cyhoeddwyd: (2010)
• PKC phosphorylates GluA1-Ser831 to enhance AMPA receptor conductance
  gan: Jenkins, Meagan A., et al.
  Cyhoeddwyd: (2012)