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AKAP79 Selectively Enhances Protein Kinase C Regulation of GluR1 at a Ca(2+)-Calmodulin-dependent Protein Kinase II/Protein Kinase C Site
Enhancement of AMPA receptor activity in response to synaptic plasticity inducing stimuli may arise, in part, through phosphorylation of the GluR1 AMPA receptor subunit at Ser-831. This site is a substrate for both Ca(2+)-calmodulin-dependent protein kinase II (CaMKII) and protein kinase C (PKC). Ho...
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| Autor principal: | |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Biochemistry and Molecular Biology
2008
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2431053/ https://ncbi.nlm.nih.gov/pubmed/18305116 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M709253200 |
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