AKAP79 Selectively Enhances Protein Kinase C Regulation of GluR1 at a Ca(2+)-Calmodulin-dependent Protein Kinase II/Protein Kinase C Site

Ítems similars

• Ca(2+)/Calmodulin-dependent Protein Kinase II Inhibitors Disrupt AKAP79-dependent PKC Signaling to GluA1 AMPA Receptors
  per: Brooks, Ian M., et al.
  Publicat: (2011)
• Regulation of GluR1 by the A-Kinase Anchoring Protein 79 (AKAP79) Signaling Complex Shares Properties with Long-Term Depression
  per: Tavalin, Steven J., et al.
  Publicat: (2002)
• Ca(2+)/Calmodulin-dependent Protein Kinase II Binds to and Phosphorylates a Specific SAP97 Splice Variant to Disrupt Association with AKAP79/150 and Modulate α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid-type Glutamate Receptor (AMPAR) Activity
  per: Nikandrova, Yelyzaveta A., et al.
  Publicat: (2010)
• Preferential generation of Ca(2+)-permeable AMPA receptors by AKAP79-anchored protein kinase C proceeds via GluA1 subunit phosphorylation at Ser-831
  per: Summers, Kyle C., et al.
  Publicat: (2019)
• Epac and Phospholipase Cε Regulate Ca(2+) Release in the Heart by Activation of Protein Kinase Cε and Calcium-Calmodulin Kinase II
  per: Oestreich, Emily A., et al.
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