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Protein Kinase Cβ2 inhibition reduces hyperglycemia-induced neural tube defects through suppression of a caspase8-triggered apoptotic pathway
BACKGROUND: Neural tube defects (NTDs) in infants of diabetic mothers are associated with increased protein kinase C β2 (PKCβ2) activity and programmed cell death (apoptosis) in the neuroepithelium during early embryogenesis. Apoptosis in diabetic embryopathy is triggered by caspase8, an initiator c...
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| Glavni autori: | , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2011
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3057385/ https://ncbi.nlm.nih.gov/pubmed/21376163 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ajog.2011.01.013 |
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