Wird geladen...

Adenosine A(2A) receptor activation attenuates tubuloglomerular feedback responses by stimulation of endothelial nitric oxide synthase

Adenosine A(2) receptors have been suggested to modulate tubuloglomerular feedback (TGF) responses by counteracting adenosine A(1) receptor-mediated vasoconstriction, but the mechanisms are unclear. We tested the hypothesis that A(2A) receptor activation blunts TGF by release of nitric oxide in the...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Hauptverfasser: Carlström, Mattias, Wilcox, Christopher S., Welch, William J.
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Physiological Society 2011
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3044007/
https://ncbi.nlm.nih.gov/pubmed/21106859
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00567.2010
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!