טוען...
Adenosine A(2A) receptor activation attenuates tubuloglomerular feedback responses by stimulation of endothelial nitric oxide synthase
Adenosine A(2) receptors have been suggested to modulate tubuloglomerular feedback (TGF) responses by counteracting adenosine A(1) receptor-mediated vasoconstriction, but the mechanisms are unclear. We tested the hypothesis that A(2A) receptor activation blunts TGF by release of nitric oxide in the...
שמור ב:
| Main Authors: | , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Physiological Society
2011
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3044007/ https://ncbi.nlm.nih.gov/pubmed/21106859 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00567.2010 |
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