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Adenosine A(2A) receptor activation attenuates tubuloglomerular feedback responses by stimulation of endothelial nitric oxide synthase
Adenosine A(2) receptors have been suggested to modulate tubuloglomerular feedback (TGF) responses by counteracting adenosine A(1) receptor-mediated vasoconstriction, but the mechanisms are unclear. We tested the hypothesis that A(2A) receptor activation blunts TGF by release of nitric oxide in the...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3044007/ https://ncbi.nlm.nih.gov/pubmed/21106859 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00567.2010 |
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