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CTLA4-Ig inhibits allergic airway inflammation by a novel CD28-independent, nitric oxide synthase-dependent mechanism

The T-cell response to antigen depends upon coordinate signaling between costimulatory and inhibitory receptors. Altered function of either may underlie the pathophysiology of autoimmune and/or chronic inflammatory diseases and manipulation of these pathways is an important emerging area of therapeu...

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Bibliographic Details
Main Authors: Deppong, Christine M., Parulekar, Amit, Boomer, Jonathan S., Bricker, Traci L., Green, Jonathan M.
Format: Artigo
Language:Inglês
Published: 2010
Subjects:
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC3039706/
https://ncbi.nlm.nih.gov/pubmed/20443189
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/eji.200940282
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