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Mice lacking Gad2 show altered behavioral effects of ethanol, flurazepam and gabaxadol
γ-Aminobutyric acid (GABA) is synthesized in brain by two isoforms of glutamic acid decarboxylase (Gad), Gad1 and Gad2. Gad1 provides most of the GABA in brain, but Gad2 can be rapidly activated in times of high GABA demand. Mice lacking Gad2 are viable whereas deletion of Gad1 is lethal. We produce...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3038569/ https://ncbi.nlm.nih.gov/pubmed/20002022 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1369-1600.2009.00186.x |
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