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Rad52 inactivation is synthetically lethal with BRCA2 deficiency
Synthetic lethality is a powerful approach to study selective cell killing based on genotype. We show that loss of Rad52 function is synthetically lethal with breast cancer 2, early onset (BRCA2) deficiency, whereas there was no impact on cell growth and viability in BRCA2-complemented cells. The fr...
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| Glavni autori: | , , , , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
National Academy of Sciences
2011
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3021033/ https://ncbi.nlm.nih.gov/pubmed/21148102 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1010959107 |
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