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Loss of the AE3 Anion Exchanger in a Hypertrophic Cardiomyopathy Model Causes Rapid Decompensation and Heart Failure
The AE3 Cl(−)/HCO(3)(−) exchanger is abundantly expressed in the sarcolemma of cardiomyocytes, where it mediates Cl(−)-uptake and HCO(3)(−)-extrusion. Inhibition of AE3-mediated Cl(−)/HCO(3)(−) exchange has been suggested to protect against cardiac hypertrophy; however, other studies indicate that A...
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| 主要な著者: | , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2010
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3019292/ https://ncbi.nlm.nih.gov/pubmed/21056571 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2010.10.028 |
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