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Zinc Overload Enhances APP Cleavage and Aβ Deposition in the Alzheimer Mouse Brain
BACKGROUND: Abnormal zinc homeostasis is involved in β-amyloid (Aβ) plaque formation and, therefore, the zinc load is a contributing factor in Alzheimer's disease (AD). However, the involvement of zinc in amyloid precursor protein (APP) processing and Aβ deposition has not been well established...
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| Autors principals: | , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Public Library of Science
2010
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3003690/ https://ncbi.nlm.nih.gov/pubmed/21179415 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0015349 |
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