Á lódáil...
Zinc Overload Enhances APP Cleavage and Aβ Deposition in the Alzheimer Mouse Brain
BACKGROUND: Abnormal zinc homeostasis is involved in β-amyloid (Aβ) plaque formation and, therefore, the zinc load is a contributing factor in Alzheimer's disease (AD). However, the involvement of zinc in amyloid precursor protein (APP) processing and Aβ deposition has not been well established...
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| Main Authors: | , , , , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Public Library of Science
2010
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3003690/ https://ncbi.nlm.nih.gov/pubmed/21179415 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0015349 |
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