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Lack of EP4 receptors on bone marrow-derived cells enhances inflammation in atherosclerotic lesions
AIM: Prostaglandin E(2), by ligation of its receptor EP4, suppresses the production of inflammatory cytokines and chemokines in macrophages in vitro. Thus, activation of EP4 may constitute an endogenous anti-inflammatory pathway. This study investigated the role of EP4 in atherosclerosis in vivo, an...
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| Autors principals: | , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Oxford University Press
2011
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3002867/ https://ncbi.nlm.nih.gov/pubmed/20736236 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvq262 |
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