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Deletion of EP4 on bone marrow–derived cells enhances inflammation and angiotensin II-induced abdominal aortic aneurysm formation
OBJECTIVE: To examine whether a lack of prostaglandin E receptor 4 (EP4) on bone marrow-derived cells would increase local inflammation and enhance the formation of abdominal aortic aneurysm (AAA) in vivo. METHODS AND RESULTS: Prostaglandin E(2) (PGE(2)), through activation of its receptor EP4, can...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3025710/ https://ncbi.nlm.nih.gov/pubmed/21088251 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.110.216580 |
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