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Impaired Orai1-mediated Resting Ca(2+) Entry Reduces the Cytosolic [Ca(2+)] and Sarcoplasmic Reticulum Ca(2+) Loading in Quiescent Junctophilin 1 Knock-out Myotubes
In the absence of store depletion, plasmalemmal Ca(2+) permeability in resting muscle is very low, and its contribution in the maintenance of Ca(2+) homeostasis at rest has not been studied in detail. Junctophilin 1 knock-out myotubes (JP1 KO) have a severe reduction in store-operated Ca(2+) entry,...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Biochemistry and Molecular Biology
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2998103/ https://ncbi.nlm.nih.gov/pubmed/20937810 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M110.149690 |
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