Modulation of experimental autoimmune encephalomyelitis through TRAF3-mediated suppression of interleukin 17 receptor signaling

Interleukin 17 (IL-17) plays critical roles in the pathogenesis of various autoimmune diseases, including experimental autoimmune encephalomyelitis (EAE). How the signals triggered by this powerful inflammatory cytokine are controlled to avoid abnormal inflammatory responses is not well understood....

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Detaylı Bibliyografya
Asıl Yazarlar: Zhu, Shu, Pan, Wen, Shi, Peiqing, Gao, Hanchao, Zhao, Fang, Song, Xinyang, Liu, Yan, Zhao, Lihua, Li, Xiaoxia, Shi, Yufang, Qian, Youcun
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: The Rockefeller University Press 2010
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Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2989772/
https://ncbi.nlm.nih.gov/pubmed/21078888
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20100703
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