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Cep152 acts as a scaffold for recruitment of Plk4 and CPAP to the centrosome
Both gain and loss of function studies have identified the Polo-like kinase Plk4/Sak as a crucial regulator of centriole biogenesis, but the mechanisms governing centrosome duplication are incompletely understood. In this study, we show that the pericentriolar material protein, Cep152, interacts wit...
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| Hauptverfasser: | , , , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
The Rockefeller University Press
2010
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2983070/ https://ncbi.nlm.nih.gov/pubmed/21059844 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.201007107 |
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