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Interleukin-17 Is Not Required for Classical Macrophage Activation in a Pulmonary Mouse Model of Cryptococcus neoformans Infection

Cryptococcus neoformans is an opportunistic fungal pathogen that causes disease in individuals with suppressed cell-mediated immunity. Recent studies in our laboratory have shown that increases in pulmonary Th1-type and interleukin-17A (IL-17A) cytokine production, classical macrophage activation, a...

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Detalles Bibliográficos
Autores principales: Hardison, Sarah E., Wozniak, Karen L., Kolls, Jay K., Wormley, Floyd L.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Microbiology (ASM) 2010
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2981312/
https://ncbi.nlm.nih.gov/pubmed/20921149
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.00845-10
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