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Genetics and complement in atypical HUS
Central to the pathogenesis of atypical hemolytic uremic syndrome (aHUS) is over-activation of the alternative pathway of complement. Following the initial discovery of mutations in the complement regulatory protein, factor H, mutations have been described in factor I, membrane cofactor protein and...
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| Main Authors: | , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Springer-Verlag
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2962786/ https://ncbi.nlm.nih.gov/pubmed/20526633 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00467-010-1555-5 |
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