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Genetics and complement in atypical HUS

Central to the pathogenesis of atypical hemolytic uremic syndrome (aHUS) is over-activation of the alternative pathway of complement. Following the initial discovery of mutations in the complement regulatory protein, factor H, mutations have been described in factor I, membrane cofactor protein and...

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Détails bibliographiques
Auteurs principaux: Kavanagh, David, Goodship, Tim
Format: Artigo
Langue:Inglês
Publié: Springer-Verlag 2010
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2962786/
https://ncbi.nlm.nih.gov/pubmed/20526633
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00467-010-1555-5
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