Sam68 regulates EMT through alternative splicing–activated nonsense-mediated mRNA decay of the SF2/ASF proto-oncogene

Epithelial-to-mesenchymal transition (EMT) and its reversal (MET) are crucial cell plasticity programs that act during development and tumor metastasis. We have previously shown that the splicing factor and proto-oncogene SF2/ASF impacts EMT/MET through production of a constitutively active splice v...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Valacca, Cristina, Bonomi, Serena, Buratti, Emanuele, Pedrotti, Simona, Baralle, Francisco Ernesto, Sette, Claudio, Ghigna, Claudia, Biamonti, Giuseppe
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: The Rockefeller University Press 2010
Θέματα:
Research Articles
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2953442/
https://ncbi.nlm.nih.gov/pubmed/20876280
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.201001073
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