Myocardin-Related Transcription Factor A Is a Common Mediator of Mechanical Stress- and Neurohumoral Stimulation-Induced Cardiac Hypertrophic Signaling Leading to Activation of Brain Natriuretic Peptide Gene Expression

Lignende værker

• Cardiac natriuretic peptides inhibit TRPC6-mediated prohypertrophic signaling through cGMP-PKG pathway
  af: Kinoshita, Hideyuki, et al.
  Udgivet: (2009)
• Reciprocal expression of MRTF-A and myocardin is crucial for pathological vascular remodelling in mice
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  Udgivet: (2012)
• O-glycosylated pro-B-type natriuretic peptide is cleaved intracellularly by furin in ventricular and atrial myocytes: importance of the distance between the O-glycosylation and cleavage sites
  af: Nishikimi, Toshio, et al.
  Udgivet: (2013)
• The effects of super-flux (high performance) dialyzer on the plasma glycosylated pro-B-type natriuretic peptide (proBNP) and glycosylated N-terminal proBNP in end-stage renal disease patients on dialysis
  af: Nakagawa, Yasuaki, et al.
  Udgivet: (2013)
• Angiotensin II type 1a receptor signalling directly contributes to the increased arrhythmogenicity in cardiac hypertrophy
  af: Yasuno, Shinji, et al.
  Udgivet: (2013)