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GRIM-19 and p16(INK4a) Synergistically Regulate Cell Cycle Progression and E2F1-responsive Gene Expression
GRIM-19 (Gene associated with Retinoid-IFN-induced Mortality-19) was originally isolated as a growth suppressor in a genome-wide knockdown screen with antisense libraries. Like classical tumor suppressors, mutations, and/or loss of GRIM-19 expression occur in primary human tumors; and it is inactiva...
Αποθηκεύτηκε σε:
| Κύριοι συγγραφείς: | , , , , , , |
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| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
American Society for Biochemistry and Molecular Biology
2010
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2934621/ https://ncbi.nlm.nih.gov/pubmed/20522552 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M110.105767 |
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