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Custom-designed proteins as novel therapeutic tools? The case of arrestins
Multiple genetic disorders can be associated with excessive signalling by mutant G-protein-coupled receptors (GPCRs) that are either constitutively active or have lost sites where phosphorylation by GPCR kinases is necessary for desensitisation by cognate arrestins. Phosphorylation-independent arres...
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| Main Authors: | , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2933791/ https://ncbi.nlm.nih.gov/pubmed/20412604 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1017/S1462399410001444 |
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