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Calmodulin kinase II, sarcoplasmic reticulum Ca(2+) leak, and atrial fibrillation
Although it is generally accepted that excitation-contraction coupling is defective in patients with atrial fibrillation, the underlying cellular mechanisms remain incompletely understood. Recent studies suggest that abnormal sarcoplasmic reticulum calcium ‘leak’ via ryanodine receptors contributes...
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| Hoofdauteurs: | , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2010
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2917388/ https://ncbi.nlm.nih.gov/pubmed/20685575 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.tcm.2010.03.004 |
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