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GSK-3β regulates post myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo
RATIONALE: Numerous studies have proposed that glycogen synthase kinase-3β (GSK-3β) is a central regulator of the hypertrophic response of cardiomyocytes. However, all of this work has relied on over-expression of GSK-3β, expression of constitutively active mutants, or small molecule inhibitors with...
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| Autors principals: | , , , , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2010
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2895414/ https://ncbi.nlm.nih.gov/pubmed/20360256 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.109.211482 |
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