GSK-3β regulates post myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo

RATIONALE: Numerous studies have proposed that glycogen synthase kinase-3β (GSK-3β) is a central regulator of the hypertrophic response of cardiomyocytes. However, all of this work has relied on over-expression of GSK-3β, expression of constitutively active mutants, or small molecule inhibitors with...

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Detalhes bibliográficos
Main Authors: Woulfe, Kathleen C., Gao, Erhe, Lal, Hind, Harris, David, Fan, Qian, Vagnozzi, Ronald, DeCaul, Morgan, Shang, Xiying, Patel, Satish, Woodgett, James R., Force, Thomas, Zhou, Jibin
Formato: Artigo
Idioma:Inglês
Publicado em: 2010
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2895414/
https://ncbi.nlm.nih.gov/pubmed/20360256
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.109.211482
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