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Basic Fibroblast Growth Factor Stimulates Matrix Metalloproteinase-13 via the Molecular Cross-talk between the Mitogen-activated Protein Kinases and Protein Kinase Cδ Pathways in Human Adult Articular Chondrocytes

Excessive release of basic fibroblast growth factor (bFGF) during loading and/or injury of the cartilage matrix may contribute to the onset or progression of osteoarthritis. This pathological role may be related to the ability of bFGF to decrease proteoglycan synthesis and to antagonize the activity...

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Autori principali: Im, Hee-Jeong, Muddasani, Prasuna, Natarajan, Viswanathan, Schmid, Thomas M., Block, Joel A., Davis, Francesca, van Wijnen, Andre J., Loeser, Richard F.
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2007
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2895271/
https://ncbi.nlm.nih.gov/pubmed/17311929
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M609040200
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