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Suppressed Accumulation of Cerebral Amyloid β Peptides in Aged Transgenic Alzheimer’s Disease Mice by Transplantation with Wild-Type or Prostaglandin E(2) Receptor Subtype 2-Null Bone Marrow

A complex therapeutic challenge for Alzheimer’s disease (AD) is minimizing deleterious aspects of microglial activation while maximizing beneficial actions, including phagocytosis/clearance of amyloid β (Aβ) peptides. One potential target is selective suppression of microglial prostaglandin E(2) rec...

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Detaylı Bibliyografya
Asıl Yazarlar: Keene, C. Dirk, Chang, Rubens C., Lopez-Yglesias, Americo H., Shalloway, Bryan R., Sokal, Izabella, Li, Xianwu, Reed, Patrick J., Keene, Lisa M., Montine, Kathleen S., Breyer, Richard M., Rockhill, Jason K., Montine, Thomas J.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Investigative Pathology 2010
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2893677/
https://ncbi.nlm.nih.gov/pubmed/20522650
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2010.090840
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