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PIB binding in aged primate brain: Enrichment of high-affinity sites in humans with Alzheimer’s disease

Aged nonhuman primates accumulate large amounts of human-sequence amyloid β (Aβ) in the brain, yet they do not manifest the full phenotype of Alzheimer’s disease (AD). To assess the biophysical properties of Aβ that might govern its pathogenic potential in humans and nonhuman primates, we incubated...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: ROSEN, REBECCA F., WALKER, LARY C., LEVINE, HARRY
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 2009
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC2891164/
https://ncbi.nlm.nih.gov/pubmed/19329226
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neurobiolaging.2009.02.011
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