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Mutations in the SH3 domain of the src oncogene which decrease association of phosphatidylinositol 3'-kinase activity with pp60v-src and alter cellular morphology.

To analyze the signaling pathways utilized in malignant transformation by pp60v-src, we have isolated and characterized src mutants which possess normal levels of protein tyrosine kinase activity but which cause only a partially transformed phenotype. Our hypothesis is that such mutants are partiall...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Wages, D S, Keefer, J, Rall, T B, Weber, M J
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 1992
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC288973/
https://ncbi.nlm.nih.gov/pubmed/1312609
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