Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor α and disuse

Παρόμοια τεκμήρια

• Analysis of multiple bone responses to graded strains above functional levels, and to disuse, in mice in vivo show that the human Lrp5 G171V High Bone Mass mutation increases the osteogenic response to loading but that lack of Lrp5 activity reduces it
  ανά: Saxon, Leanne K., κ.ά.
  Έκδοση: (2011)
• The mouse fibula as a suitable bone for the study of functional adaptation to mechanical loading
  ανά: Moustafa, Alaa, κ.ά.
  Έκδοση: (2009)
• Loading-related Regulation of Transcription Factor EGR2/Krox-20 in Bone Cells Is ERK1/2 Protein-mediated and Prostaglandin, Wnt Signaling Pathway-, and Insulin-like Growth Factor-I Axis-dependent
  ανά: Zaman, Gul, κ.ά.
  Έκδοση: (2012)
• Lrp5 Is Not Required for the Proliferative Response of Osteoblasts to Strain but Regulates Proliferation and Apoptosis in a Cell Autonomous Manner
  ανά: Javaheri, Behzad, κ.ά.
  Έκδοση: (2012)
• Mechano-transduction in Osteoblastic Cells Involves Strain-regulated Estrogen Receptor α-mediated Control of Insulin-like Growth Factor (IGF) I Receptor Sensitivity to Ambient IGF, Leading to Phosphatidylinositol 3-Kinase/AKT-dependent Wnt/LRP5 Receptor-independent Activation of β-Catenin Signaling
  ανά: Sunters, Andrew, κ.ά.
  Έκδοση: (2010)