טוען...
Polycystin-2 Activation by Inositol 1,4,5-Trisphosphate-induced Ca(2+) Release Requires Its Direct Association with the Inositol 1,4,5-Trisphosphate Receptor in a Signaling Microdomain
Autosomal dominant polycystic kidney disease is characterized by the loss-of-function of a signaling complex involving polycystin-1 and polycystin-2 (TRPP2, an ion channel of the TRP superfamily), resulting in a disturbance in intracellular Ca(2+) signaling. Here, we identified the molecular determi...
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| Main Authors: | , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Biochemistry and Molecular Biology
2010
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| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2881802/ https://ncbi.nlm.nih.gov/pubmed/20375013 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M109.090662 |
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