Iron sequestration by macrophages decreases the potential for extracellular hydroxyl radical formation.

Podobne knjige/članki

• The Nature of Extracellular Iron Influences Iron Acquisition by Mycobacterium tuberculosis Residing within Human Macrophages
  od: Olakanmi, Oyebode, et al.
  Izdano: (2004)
• Protease-cleaved iron-transferrin augments oxidant-mediated endothelial cell injury via hydroxyl radical formation.
  od: Miller, R A, et al.
  Izdano: (1995)
• Neutrophil degranulation inhibits potential hydroxyl-radical formation. Relative impact of myeloperoxidase and lactoferrin release on hydroxyl-radical production by iron-supplemented neutrophils assessed by spin-trapping techniques.
  od: Britigan, B E, et al.
  Izdano: (1989)
• Protease cleavage of iron-transferrin augments pyocyanin-mediated endothelial cell injury via promotion of hydroxyl radical formation.
  od: Miller, R A, et al.
  Izdano: (1996)
• Pseudomonas and neutrophil products modify transferrin and lactoferrin to create conditions that favor hydroxyl radical formation.
  od: Britigan, B E, et al.
  Izdano: (1991)