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Antigen-Specific CD25(−)Foxp3(−)IFN-γ(high)CD4(+) T Cells Restrain the Development of Experimental Allergic Encephalomyelitis by Suppressing Th17
The current study identifies within the Th1 subtype two distinct CD4(+) populations: those capable of transferring inflammatory autoimmunity and others that regulate its development by suppressing Th17 in an interferon (IFN)-γ-dependent manner. These CD4(+)IFN-γ(high)IL-4(low)IL-10(low)TGF-β(low)FOX...
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| Autori principali: | , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Society for Investigative Pathology
2010
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2877838/ https://ncbi.nlm.nih.gov/pubmed/20382706 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2010.090855 |
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