An Autoinflammatory Disease with Deficiency of the Interleukin-1–Receptor Antagonist

BACKGROUND: Autoinflammatory diseases manifest inflammation without evidence of infection, high-titer autoantibodies, or autoreactive T cells. We report a disorder caused by mutations of IL1RN, which encodes the interleukin-1–receptor antagonist, with prominent involvement of skin and bone. METHODS:...

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Detalhes bibliográficos
Principais autores: Aksentijevich, Ivona, Masters, Seth L., Ferguson, Polly J., Dancey, Paul, Frenkel, Joost, van Royen-Kerkhoff, Annet, Laxer, Ron, Tedgård, Ulf, Cowen, Edward W., Pham, Tuyet-Hang, Booty, Matthew, Estes, Jacob D., Sandler, Netanya G., Plass, Nicole, Stone, Deborah L., Turner, Maria L., Hill, Suvimol, Butman, John A., Schneider, Rayfel, Babyn, Paul, El-Shanti, Hatem I., Pope, Elena, Barron, Karyl, Bing, Xinyu, Laurence, Arian, Lee, Chyi-Chia R., Chapelle, Dawn, Clarke, Gillian I., Ohson, Kamal, Nicholson, Marc, Gadina, Massimo, Yang, Barbara, Korman, Benjamin D., Gregersen, Peter K., van Hagen, P. Martin, Hak, A. Elisabeth, Huizing, Marjan, Rahman, Proton, Douek, Daniel C., Remmers, Elaine F., Kastner, Daniel L., Goldbach-Mansky, Raphaela
Formato: Artigo
Idioma:Inglês
Publicado em: 2009
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2876877/
https://ncbi.nlm.nih.gov/pubmed/19494218
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1056/NEJMoa0807865
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