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An Autoinflammatory Disease with Deficiency of the Interleukin-1–Receptor Antagonist
BACKGROUND: Autoinflammatory diseases manifest inflammation without evidence of infection, high-titer autoantibodies, or autoreactive T cells. We report a disorder caused by mutations of IL1RN, which encodes the interleukin-1–receptor antagonist, with prominent involvement of skin and bone. METHODS:...
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| 主要な著者: | , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2009
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2876877/ https://ncbi.nlm.nih.gov/pubmed/19494218 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1056/NEJMoa0807865 |
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