Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.

Herpes simplex virus infection of mammalian hosts involves lytic replication at a primary site, such as the cornea, translocation by axonal transport to sensory ganglia and replication, and latent infection at a secondary site, ganglionic neurons. The virus-encoded thymidine kinase, which is a targe...

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Detaylı Bibliyografya
Yayımlandı:Proc Natl Acad Sci U S A
Asıl Yazarlar: Coen, D M, Kosz-Vnenchak, M, Jacobson, J G, Leib, D A, Bogard, C L, Schaffer, P A, Tyler, K L, Knipe, D M
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: National Academy of Sciences 1989
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC287348/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2543985/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.86.12.4736
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