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Calcium and ER stress mediate hepatic apoptosis after burn injury

A hallmark of the disease state following severe burn injury is decreased liver function, which results in gross metabolic derangements that compromise patient survival. The underlying mechanisms leading to hepatocyte dysfunction after burn are essentially unknown. The aim of the present study was t...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Jeschke, Marc G., Gauglitz, Gerd G., Song, Juquan, Kulp, Gabriela A., Finnerty, Celeste C., Cox, Robert A., Barral, José M., Herndon, David N., Boehning, Darren
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: Blackwell Publishing Ltd 2009
Θέματα:
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2855735/
https://ncbi.nlm.nih.gov/pubmed/20141609
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1582-4934.2008.00644.x
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