β-Amyloid(1–42) Gene Transfer Model Exhibits Intraneuronal Amyloid, Gliosis, Tau Phosphorylation, and Neuronal Loss

Ítems similars

• Fractalkine signaling and Tau hyper-phosphorylation are associated with autophagic alterations in lentiviral Tau and Aβ(1–42) gene transfer models
  per: Hebron, Michaeline L., et al.
  Publicat: (2013)
• Tau deletion impairs intracellular β-amyloid-42 clearance and leads to more extracellular plaque deposition in gene transfer models
  per: Lonskaya, Irina, et al.
  Publicat: (2014)
• Human APOE genotype affects intraneuronal Aβ(1–42) accumulation in a lentiviral gene transfer model
  per: Zhao, Wenjuan, et al.
  Publicat: (2014)
• Apolipoprotein E decreases tau kinases and phospho-tau levels in primary neurons
  per: Hoe, Hyang-Sook, et al.
  Publicat: (2006)
• Do cerebrospinal fluid transfer methods affect measured amyloid β42, total tau, and phosphorylated tau in clinical practice?
  per: Paterson, Ross W., et al.
  Publicat: (2015)