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Disruption of the K(+) Channel β-Subunit KCNE3 Reveals an Important Role in Intestinal and Tracheal Cl(−) Transport
The KCNE3 β-subunit constitutively opens outwardly rectifying KCNQ1 (Kv7.1) K(+) channels by abolishing their voltage-dependent gating. The resulting KCNQ1/KCNE3 heteromers display enhanced sensitivity to K(+) channel inhibitors like chromanol 293B. KCNE3 was also suggested to modify biophysical pro...
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| 主要な著者: | , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Biochemistry and Molecular Biology
2010
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2844166/ https://ncbi.nlm.nih.gov/pubmed/20051516 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M109.047829 |
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