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NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa
Macula densa (MD)-mediated regulation of renal hemodynamics via tubuloglomerular feedback is regulated by interactions between factors such as superoxide (O(2)(−)) and angiotensin II (ANG II). We have reported that NaCl-induced O(2)(−) in the MD is produced by the NOX2 isoform of NADPH oxidase (NOX)...
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| Hoofdauteurs: | , , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Physiological Society
2010
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2838666/ https://ncbi.nlm.nih.gov/pubmed/20053956 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00762.2009 |
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