TNF dependency of IL-17-induced joint pathology can be circumvented by Toll-like receptor-2 signaling

Ítems similars

• IL-1-dependent cartilage damage in a macrophage-driven arthritis model can be circumvented by T cell IL-17
  per: Koenders, MI, et al.
  Publicat: (2004)
• TNF-α dependency of IL-17-induced joint pathology differs under naive and arthritis conditions in vivo
  per: Koenders, M, et al.
  Publicat: (2003)
• Neutralizing IL-17 during re-activation of experimental arthritis prevents joint inflammation and bone erosion by decreasing RANKL and IL-1
  per: Koenders, MI, et al.
  Publicat: (2005)
• IL-17 derived from juxta-articular bone and synovium contributes to joint degradation in rheumatoid arthritis
  per: Chabaud, Martine, et al.
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• IL-17 produced by Paneth cells drives TNF-induced shock
  per: Takahashi, Nozomi, et al.
  Publicat: (2008)