TNF-α dependency of IL-17-induced joint pathology differs under naive and arthritis conditions in vivo

Ítems similars

• IL-1-dependent cartilage damage in a macrophage-driven arthritis model can be circumvented by T cell IL-17
  per: Koenders, MI, et al.
  Publicat: (2004)
• Local IL-17 gene therapy accelerates collagen arthritis with severe bone erosion and rank ligand and rank expression in synovial infiltrate and at bone erosion sites
  per: Lubberts, E, et al.
  Publicat: (2001)
• Neutralizing IL-17 during re-activation of experimental arthritis prevents joint inflammation and bone erosion by decreasing RANKL and IL-1
  per: Koenders, MI, et al.
  Publicat: (2005)
• TNF dependency of IL-17-induced joint pathology can be circumvented by Toll-like receptor-2 signaling
  per: Van den Berg, W, et al.
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• IL-18 expression in synovial biopsies of patients with active rheumatoid arthritis is associated with enhanced levels of both IL-1 and TNFα
  per: Joosten, LAB, et al.
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