TNF dependency of IL-17-induced joint pathology can be circumvented by Toll-like receptor-2 signaling

Samankaltaisia teoksia

• IL-1-dependent cartilage damage in a macrophage-driven arthritis model can be circumvented by T cell IL-17
  Tekijä: Koenders, MI, et al.
  Julkaistu: (2004)
• TNF-α dependency of IL-17-induced joint pathology differs under naive and arthritis conditions in vivo
  Tekijä: Koenders, M, et al.
  Julkaistu: (2003)
• Neutralizing IL-17 during re-activation of experimental arthritis prevents joint inflammation and bone erosion by decreasing RANKL and IL-1
  Tekijä: Koenders, MI, et al.
  Julkaistu: (2005)
• IL-17 derived from juxta-articular bone and synovium contributes to joint degradation in rheumatoid arthritis
  Tekijä: Chabaud, Martine, et al.
  Julkaistu: (2001)
• IL-17 produced by Paneth cells drives TNF-induced shock
  Tekijä: Takahashi, Nozomi, et al.
  Julkaistu: (2008)