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HUCB Cell Therapy Blocks the Morphological Change and Recruitment of CD11b-Expressing, Isolectin-Binding Proinflammatory Cells after MCAO
Secondary neurodegeneration resulting from stroke is mediated by delayed proinflammatory signaling and immune cell activation. Although it remains unknown which cell surface markers signify a proinflammatory phenotype, increased isolectin binding occurs on CD11b-expressing immune cells within injure...
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| Hauptverfasser: | , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2010
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2830323/ https://ncbi.nlm.nih.gov/pubmed/19998484 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jnr.22306 |
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