טוען...
Epileptogenesis causes an N-methyl-d-aspartate receptor/Ca(2+)-dependent decrease in Ca(2+)/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent epileptiform discharges
Alterations in the function of Ca(2+)/calmodulin-dependent protein kinase II (CaM Kinase II) have been observed in both in vivo and in vitro models of epileptogenesis; however the molecular mechanism mediating the effects of epileptogenesis on CaM Kinase II have not been elucidated. This study was i...
שמור ב:
| Main Authors: | , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2008
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2819419/ https://ncbi.nlm.nih.gov/pubmed/18495112 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ejphar.2008.04.021 |
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