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Thalidomide attenuates nitric oxide-driven angiogenesis by interacting with soluble guanylyl cyclase

BACKGROUND AND PURPOSE: Nitric oxide (NO) promotes angiogenesis by activating endothelial cells. Thalidomide arrests angiogenesis by interacting with the NO pathway, but its putative targets are not known. Here, we have attempted to identify these targets. EXPERIMENTAL APPROACH: Cell-based angiogene...

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Detaylı Bibliyografya
Asıl Yazarlar: Majumder, Syamantak, Rajaram, Megha, Muley, Ajit, Reddy, Himabindu S, Tamilarasan, KP, Kolluru, Gopi Krishna, Sinha, Swaraj, Siamwala, Jamila H, Gupta, Ravi, Ilavarasan, R, Venkataraman, S, Sivakumar, KC, Anishetty, Sharmila, Kumar, Pradeep G, Chatterjee, Suvro
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Blackwell Publishing Ltd 2009
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2801213/
https://ncbi.nlm.nih.gov/pubmed/19912234
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1476-5381.2009.00446.x
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