Thalidomide attenuates nitric oxide-driven angiogenesis by interacting with soluble guanylyl cyclase

BACKGROUND AND PURPOSE: Nitric oxide (NO) promotes angiogenesis by activating endothelial cells. Thalidomide arrests angiogenesis by interacting with the NO pathway, but its putative targets are not known. Here, we have attempted to identify these targets. EXPERIMENTAL APPROACH: Cell-based angiogene...

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Detalhes bibliográficos
Main Authors: Majumder, Syamantak, Rajaram, Megha, Muley, Ajit, Reddy, Himabindu S, Tamilarasan, KP, Kolluru, Gopi Krishna, Sinha, Swaraj, Siamwala, Jamila H, Gupta, Ravi, Ilavarasan, R, Venkataraman, S, Sivakumar, KC, Anishetty, Sharmila, Kumar, Pradeep G, Chatterjee, Suvro
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Publishing Ltd 2009
Assuntos:
Research Papers
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2801213/
https://ncbi.nlm.nih.gov/pubmed/19912234
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1476-5381.2009.00446.x
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