Thalidomide attenuates nitric oxide-driven angiogenesis by interacting with soluble guanylyl cyclase

BACKGROUND AND PURPOSE: Nitric oxide (NO) promotes angiogenesis by activating endothelial cells. Thalidomide arrests angiogenesis by interacting with the NO pathway, but its putative targets are not known. Here, we have attempted to identify these targets. EXPERIMENTAL APPROACH: Cell-based angiogene...

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Autors principals: Majumder, Syamantak, Rajaram, Megha, Muley, Ajit, Reddy, Himabindu S, Tamilarasan, KP, Kolluru, Gopi Krishna, Sinha, Swaraj, Siamwala, Jamila H, Gupta, Ravi, Ilavarasan, R, Venkataraman, S, Sivakumar, KC, Anishetty, Sharmila, Kumar, Pradeep G, Chatterjee, Suvro
Format: Artigo
Idioma:Inglês
Publicat: Blackwell Publishing Ltd 2009
Matèries:
Research Papers
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2801213/
https://ncbi.nlm.nih.gov/pubmed/19912234
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1476-5381.2009.00446.x
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