טוען...
Inhibition of Protein Kinase CK2 Closes the CFTR Cl(−) Channel, but has no Effect on the Cystic Fibrosis Mutant ΔF508-CFTR
BACKGROUND: Deletion of phenylalanine-508 (ΔF508) from the first nucleotide-binding domain (NBD1) in the wild-type cystic fibrosis (CF) transmembrane-conductance regulator (wtCFTR) causes CF. However, the mechanistic relationship between ΔF508-CFTR and the diversity of CF disease is unexplained. The...
שמור ב:
| Main Authors: | , , , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2009
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2795324/ https://ncbi.nlm.nih.gov/pubmed/19910675 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1159/000257427 |
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