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Human RAP1 inhibits non-homologous end joining at telomeres
Telomeres, the nucleoprotein structures at the ends of linear chromosomes, promote genome stability by distinguishing chromosome termini from DNA double-strand breaks (DSBs). Cells possess two principal pathways for DSB repair: homologous recombination and non-homologous end joining (NHEJ). Several...
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| Main Authors: | , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Nature Publishing Group
2009
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2776107/ https://ncbi.nlm.nih.gov/pubmed/19763083 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/emboj.2009.275 |
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