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Paradoxical Activation of Endothelial Nitric Oxide Synthase by NADPH oxidase
OBJECTIVES: Increased formation of reactive oxygen species (ROS) has been identified as a causative factor in endothelial dysfunction by reducing NO bioavailability and uncoupling endothelial nitric oxide synthase (eNOS). However, the specific contribution of ROS to endothelial function is not well...
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| Autori principali: | , , , , , , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2008
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2771553/ https://ncbi.nlm.nih.gov/pubmed/18556569 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.108.168278 |
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