Paradoxical Activation of Endothelial Nitric Oxide Synthase by NADPH oxidase

OBJECTIVES: Increased formation of reactive oxygen species (ROS) has been identified as a causative factor in endothelial dysfunction by reducing NO bioavailability and uncoupling endothelial nitric oxide synthase (eNOS). However, the specific contribution of ROS to endothelial function is not well...

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Detaylı Bibliyografya
Asıl Yazarlar: Zhang, Qian, Malik, Pulkit, Pandey, Deepesh, Gupta, Sonali, Jagnandan, Davin, de Chantemele, Eric Belin, Banfi, Botond, Marrero, Mario B., Rudic, R Daniel, Stepp, David W., Fulton, David J.R.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2008
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2771553/
https://ncbi.nlm.nih.gov/pubmed/18556569
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.108.168278
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